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Rotenone treatment significantly reduced survival compared to untreated controls, confirming that mitochondrial respiration is a critical mediator of cholesterol-induced hepatic reprogramming and host defense ( Fig

HMGB1: an overview of its versatile roles in the pathogenesis of colorectal cancer

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the total number of actin enriched spines (red) adjacent to VGLUT1 or Synapsin were counted and converted to a percentage as the percent correlation of treatment-induced spines to presynaptic markers is a strong indicator of ability to transmit excitatory signals