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Shin JH, Park SJ, Kim ES, Jo YK, Hong J, Cho DH

Therefore, DON treatment can not only reduce the self-renewal potential and metastatic ability of pancreatic ductal adenocarcinoma (PDAC) tumor cells but also lead to extensive remodeling of the tumor extracellular matrix (ECM), promoting the infiltration and activation of CD8 + T cells (Sharma et al., 2020)

Its molecular design gives it an impressively long half-life of about seven days

The persistently elevated risk amongst these trial populations demonstrates the limitations of current management strategies and highlights the need for therapies that address the link between metabolic disease and direct cardiovascular pathology