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Research has demonstrated that glutathione depletion in liver cells can lead to increased susceptibility to damage from various hepatotoxins

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Clinical success with ACI is highly dependent on defect etiology, morphology, and location

Together, these interconnected signaling cascades reshape transcriptional programs in fibroblasts, AECs, and endothelial cells, promoting myofibroblast differentiation, epithelial cell dysfunction, immune dysregulation, and excessive ECM deposition, which collectively drive the onset and progression of pulmonary fibrosis [33, 39] (Fig