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We propose that melatonin acts as a metabolic modulator in ILC2s, redirecting glucose utilization toward redox-supporting pathways that restrain inflammatory effector programs

This view of rodent islet blood flow has long been under discussion (see [39] for the different models of islet microcirculation) and new observations utilising in vivo imaging of islet blood flow challenge even these models [40], making the issue of paracrine effects through the bloodstream for rodent islets open for debate

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Drug interactions and age-related changes in metabolism can affect how the medication works, so dosing must be personalized accordingly