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[DOI] [PMC free article] [PubMed] [Google Scholar] 103.Raub A et al

Corrected and uncorrected ETC activity is shown in Figures 5a and b, respectively, and demonstrate a partial defect in complexes I/III and I/III rotenone sensitive (RS)

Complementarily, IGF-1R preferentially activates IRS-2 [120] as it was found that IRS-2 contains a KLRB domain that functions to block the tyrosine kinase domain in the cytoplasmic region of the IR, and that such does not happen in the IGF-1R

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