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Additionally, by binding to GLP-1 receptors in enteric neurons and endocrine cells, these agents slow gastric motility, reduce acid secretion, and modulate appetite through satiety signaling to the hypothalamus [3]
Synthetic biology techniques, such as gene editing and genetic engineering, can greatly improve biological activity and deliver MOTS-c directly to the acting site, thus further expanding the therapeutic application of MOTS-c
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