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This so-called ETosis is often associated with cell death of the producing MC, and the type of stimulus potentially determines the ratio of surviving vs

Among studied drugs (Efavirenz, Tenofovir, Emtricitabine, Lamivudine, and Indinavir), only Efavirenz increased ER stress via upregulation and activation of the inositol requiring kinase 1 (IRE1) and protein kinase-like ER kinase (PERK) signaling

13, 14 The GFAP is considered a very important stress marker in diverse retinal pathologies and in experimental models of ocular hypertension