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GHK-Cu interacts with cells in ways that stimulate repair and protect against oxidative stress

Overexpression of FSP1 or treatment with Fer-1, a ferroptosis inhibitor, significantly reversed the apoptosis, ferroptosis, and lipid peroxidation induced by the activation of the MPO/HOCl signaling pathway, exerting neuroprotective effects

Migration assay data further suggested better-supported cellular motility in peptide-exposed cultures, a process linked to cytoskeletal dynamics and focal adhesion regulation

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