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Temperature excursions above 8C denature the peptide structure irreversibly, eliminating therapeutic activity
Ultimately, this review proposes that PEM may arise from a complex interplay among mitochondrial dysfunction, immune activation, and neuroinflammation, which together form a self-perpetuating loop of energy exhaustion - inflammation amplification, potentially contributing to the chronic and multi-system nature of PEM symptoms
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A hybrid machine learning method for fusing fMRI and genetic data: combining both improves classification of schizophrenia
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