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doi: 10.1056/NEJMoa1603827

Roux-en-Y gastric bypass surgery suppresses hepatic gluconeogenesis and increases intestinal gluconeogenesis in a T2DM rat model

Dempsey reported that many of her patients with gut motility issues are doing well on the low doses of the drug being used, and a mast cell paper reported that reductions in nausea were common

Combined activation of GLP-1, GIP, and GCG systems theoretically provides complementary mechanisms: GLP-1-mediated insulin sensitization and satiety promotion, GIP-mediated enhancement of insulin secretion and adipose lipid buffering, and glucagon-mediated direct hepatic lipid oxidation and energy expenditure augmentation